Available studies (27 found)

Short citationFull citationBioassayEpidemiologyEpi. meta-analysisIn vitroEcology
Biesemeier et al. 2011 Biesemeier JA et al. An oral developmental neurotoxicity study of decabromodiphenyl ether (DecaBDE) in rats. Birth Defects Res. B Dev. Reprod. Toxicol. 2011; 92 (1):17-35. PubMed DOI
Blanco et al. 2013 Blanco J et al. Perinatal exposure to BDE-99 causes learning disorders and decreases serum thyroid hormone levels and BDNF gene expression in hippocampus in rat offspring. Toxicology 2013; 308 ():122-8. PubMed DOI
Bowers et al. 2015 Bowers WJ et al. Behavioral and thyroid effects of in utero and lactational exposure of Sprague-Dawley rats to the polybrominated diphenyl ether mixture DE71. Neurotoxicol Teratol ; 52 (Pt B):127-42. PubMed DOI
Buratovic et al. 2014 Buratovic S et al. Developmental exposure to the polybrominated diphenyl ether PBDE 209: Neurobehavioural and neuroprotein analysis in adult male and female mice. Environ. Toxicol. Pharmacol. 2014; 38 (2):570-85. PubMed DOI
Chen et al. 2014 Chen YH et al. Prenatal exposure to decabrominated diphenyl ether impairs learning ability by altering neural stem cell viability, apoptosis, and differentiation in rat hippocampus. Hum Exp Toxicol 2014; ():None. PubMed
Cheng et al. 2009 Cheng J et al. Neurobehavioural effects, redox responses and tissue distribution in rat offspring developmental exposure to BDE-99. Chemosphere 2009; 75 (7):963-8. PubMed DOI
Driscoll et al. 2009 Driscoll LL, Gibson AM, and Hieb A. Chronic postnatal DE-71 exposure: effects on learning, attention and thyroxine levels. Neurotoxicol Teratol ; 31 (2):76-84. PubMed DOI
Driscoll et al. 2012 Driscoll LL et al. Acute postnatal exposure to the pentaBDE commercial mixture DE-71 at 5 or 15 mg/kg/day does not produce learning or attention deficits in rats. Neurotoxicol Teratol ; 34 (1):20-6. PubMed DOI
Dufault et al. 2005 Dufault C, Poles G, and Driscoll LL. Brief postnatal PBDE exposure alters learning and the cholinergic modulation of attention in rats. Toxicol. Sci. 2005; 88 (1):172-80. PubMed
Eriksson et al. 2001 Eriksson P, Jakobsson E, and Fredriksson A. Brominated flame retardants: a novel class of developmental neurotoxicants in our environment?. Environ. Health Perspect. 2001; 109 (9):903-8. PubMed
Fischer et al. 2008 Fischer C, Fredriksson A, and Eriksson P. Coexposure of neonatal mice to a flame retardant PBDE 99 (2,2',4,4',5-pentabromodiphenyl ether) and methyl mercury enhances developmental neurotoxic defects. Toxicol. Sci. 2008; 101 (2):275-85. PubMed
He et al. 2009 He P et al. Mechanisms underlying the developmental neurotoxic effect of PBDE-47 and the enhanced toxicity associated with its combination with PCB153 in rats. Neurotoxicology 2009; 30 (6):1088-95. PubMed DOI
He et al. 2011 He P et al. Toxic effect of PBDE-47 on thyroid development, learning, and memory, and the interaction between PBDE-47 and PCB153 that enhances toxicity in rats. Toxicol Ind Health 2011; 27 (3):279-88. PubMed DOI
Koenig et al. 2012 Koenig CM et al. Maternal transfer of BDE-47 to offspring and neurobehavioral development in C57BL/6J mice. Neurotoxicol Teratol ; 34 (6):571-80. PubMed DOI
Llansola et al. 2009 Llansola M et al. Increasing the function of the glutamate-nitric oxide-cyclic guanosine monophosphate pathway increases the ability to learn a Y-maze task. J. Neurosci. Res. 2009; 87 (10):2351-5. PubMed DOI
Reverte et al. 2013 Reverte I et al. Long term effects of murine postnatal exposure to decabromodiphenyl ether (BDE-209) on learning and memory are dependent upon APOE polymorphism and age. Neurotoxicol Teratol ; 40 ():17-27. PubMed DOI
Reverte et al. 2014 Reverte I et al. Thyroid hormones and fear learning but not anxiety are affected in adult apoE transgenic mice exposed postnatally to decabromodiphenyl ether (BDE-209). Physiol. Behav. 2014; 133 ():81-91. PubMed DOI
Rice et al. 2009 Rice DC et al. Behavioral changes in aging but not young mice after neonatal exposure to the polybrominated flame retardant decaBDE. Environ. Health Perspect. 2009; 117 (12):1903-11. PubMed DOI
Ta et al. 2011 Ta TA et al. Bioaccumulation and behavioral effects of 2,2',4,4'-tetrabromodiphenyl ether (BDE-47) in perinatally exposed mice. Neurotoxicol Teratol ; 33 (3):393-404. PubMed DOI
Verma et al. 2013 Verma, P., P. Singh, and B.S. Gandhi. 2013. Prophylactic efficacy of Bacopa monnieri on decabromodiphenyl ether (PBDE-209)-induced alterations in oxidative status and spatial memory in mice. Asian Journal of Pharmaceutical and Clinical Research 6(3):242-247
Verma et al. 2014 Verma P, Singh P, and Gandhi BS. Neuromodulatory role of Bacopa monnieri on oxidative stress induced by postnatal exposure to decabromodiphenyl ether (PBDE -209) in neonate and young female mice. Iran J Basic Med Sci 2014; 17 (4):307-11. PubMed
Viberg et al. 2003 Viberg H, Fredriksson A, and Eriksson P. Neonatal exposure to polybrominated diphenyl ether (PBDE 153) disrupts spontaneous behaviour, impairs learning and memory, and decreases hippocampal cholinergic receptors in adult mice. Toxicol. Appl. Pharmacol. 2003; 192 (2):95-106. PubMed
Viberg et al. 2006 Viberg H et al. Neonatal exposure to higher brominated diphenyl ethers, hepta-, octa-, or nonabromodiphenyl ether, impairs spontaneous behavior and learning and memory functions of adult mice. Toxicol. Sci. 2006; 92 (1):211-8. PubMed
Woods et al. 2012 Woods R et al. Long-lived epigenetic interactions between perinatal PBDE exposure and Mecp2308 mutation. Hum. Mol. Genet. 2012; 21 (11):2399-411. PubMed DOI
Zhang et al. 2013 Zhang H et al. Lactation exposure to BDE-153 damages learning and memory, disrupts spontaneous behavior and induces hippocampus neuron death in adult rats. Brain Res. 2013; 1517 ():44-56. PubMed DOI

Showing studies 1-25 of 27