Available studies (208 found)

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Short citationFull citationBioassayEpidemiologyEpi. meta-analysisIn vitroEcology
ECHA 2009c ECHA. 2009c. BPADP: Exp Key Developmental toxicity / teratogenicity.002.
ECHA 2009d ECHA. 2009d. BPS-MAE: Exp Supporting Developmental toxicity / teratogenicity.002.
ECHA 2010a ECHA. 2010a. BPADP: Exp Key Repeated dose toxicity: oral.001.
ECHA 2010b ECHA. 2010b. BPADP: Exp Key Repeated dose toxicity: oral.002.
ECHA 2010c ECHA. 2010c. (1-methylethylidene)di-4,1-phenylenetetraphenyl diphosphate: Exp Key Repeated dose toxicity: oral.002.
ECHA 2010d ECHA. 2010d. BPS-MAE: Exp Key Developmental toxicity / teratogenicity.001
ECHA 2011a ECHA. 2011a. N-(p-toluenesulfonyl)-N'-(3-(p-toluenesulfonyloxy)phenyl)urea: Exp Key Toxicity to reproduction.001.
ECHA 2011b ECHA. 2011b. (1-methylethylidene)di-4,1-phenylenetetraphenyl diphosphate: Exp Key Repeated dose toxicity: oral.001.
ECHA 2011c ECHA. 2011c. (1-methylethylidene)di-4,1-phenylenetetraphenyl diphosphate: Exp Key Repeated dose toxicity: oral.002.
ECHA 2011d ECHA. 2011d. BPS-MAE: Exp Key Toxicity to reproduction.001.
ECHA 2014a ECHA. 2014a. 4,4'-sulphonyldiphenol: Exp Key Repeated dose toxicity: oral.001.
ECHA 2014b ECHA. 2014b. 4,4'-sulphonyldiphenol: Exp Key Developmental toxicity / teratogenicity.001.
ECHA 3 ECHA. BPADP: NS NS Repeated dose toxicity: oral.009.
ECHA 4 ECHA. BPADP: NS NS Developmental toxicity / teratogenicity.004.
ECHA 5 ECHA. BPADP: NS NS Developmental toxicity / teratogenicity.005.
Eastman Kodak 1991 Eastman Kodak. 1991. Letter concerning enclosed information on bisphenol S with attachments. EPA/OTS. 920000124:#86-920000124.
Eladak et al. 2015 Eladak S, Grisin T, Moison D, Guerquin MJ, N'Tumba-Byn T, Pozzi-Gaudin S, Benachi A, Livera G, Rouiller-Fabre V, Habert R. 2015. A new chapter in the bisphenol A story: Bisphenol S and bisphenol F are not safe alternatives to this compound. Fertil Steril 103:11-21. PubMed DOI
Fang et al. 2003 Fang H, Tong W, Branham WS, Moland CL, Dial SL, Hong H, et al. 2003. Study of 202 natural, synthetic, and environmental chemicals for binding to the androgen receptor. Chem Res Toxicol 16:1338-1358. PubMed
Feng et al. 2012 Feng Y, Yin J, Jiao Z, Shi J, Li M, Shao B. 2012. Bisphenol AF may cause testosterone reduction by directly affecting testis function in adult male rats. Toxicol Lett 211(2):201-9. PubMed DOI
Fic et al. 2013 Fic A, Zegura B, Sollner Dolenc M, et al. 2013. Mutagenicity and DNA damage of bisphenol A and its structural analogues in HepG2 cells. Arh Hig Rada Toksikol 64:3-14. PubMed DOI
Fic et al. 2014 Fic A, Zegura B, Gramec D, Masic LP. 2014. Estrogenic and androgenic activities of TBBA and TBMEPH, metabolites of novel brominated flame retardants, and selected bisphenols, using the XenoScreen XL YES/YAS assay. Chemosphere 112:362-369. PubMed DOI
Furr 2014 Furr JR, Lambright CS, Wilson VS, Foster PM, Gray LE. 2014. A short-term in vivo screen using fetal testosterone production, a key event in the phthalate adverse outcome pathway, to predict disruption of sexual differentiation. Toxicol Sci 140(2):403-424. PubMed DOI
Gaido et al. 2000 Gaido KW, Maness SC, McDonnell DP, Dehal SS, Kupfer D, Safe S. 2000. Interaction of methoxychlor and related compounds with estrogen receptor alpha and beta, and androgen receptor: Structure-activity studies. Mol Pharmacol 58(4):852-8. PubMed
Gao et al. 2015 Gao X, Ma J, Chen Y, Wang HS. 2015. Rapid responses and mechanism of action for low-dose bisphenol S on rat hearts and isolated myocytes: Evidence of female-specific proarrhythmic effects. Environ Health Perspect 123:571-8. PubMed DOI
Gilbert et al. 1994 Gilbert J, Dore J-C, Bignon E, Pons M, Ojasoo T. 1994. Study of the effects of basic di- and tri-phenyl derivatives on malignant cell proliferation: An example of the application of correspondence factor analysis to structure-activity relationships (SAR). Quant Struct-Act Relat 13:262-274.

Showing studies 51-75 of 208